Erratum to vol 52, no. 4, Jan/Feb 2010

Article Outline

• Copyright

The following figures should have appeared in the color in Unusual Cardiomyopathies issue of Progress in Cardiovascular Diseases, vol 52, no. 4, Jan/Feb 2010. The publisher regrets this error. All figures are available online at www.onlinepcd.com.

O’Mahony C, Elliott P: Anderson-Fabry disease and the heart. Prog Cardiovasc Dis 2010;52:326–335.

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• Fig. 1. 

  Histologic findings in the myocardium of a patient with AFD. Hematoxylin and eosin staining showing storage (perinuclear vacuoles) and hypertrophy of cardiomyocytes. Reproduced from Linhart A, Elliott PM. The heart in Anderson-Fabry disease and other lysosomal storage disorders. Heart 93:528-535, 2007, with permission from BMJ Publishing Group Ltd.

Falk RH, Dubrey SW: Amyloid heart disease. Prog Cardiovasc Dis 2010:52:347–361.

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• Fig. 2. 

  Endomyocardial biopsy specimen of a patient with cardiac amyloidosis, stained with sulfated Alcian blue. There is extensive extracellular amyloid deposition (turquoise stain) which distorts the myocytes.

Dubrey SW, Falk RH: Diagnosis and management of cardiac sarcoidosis. Prog Cardiovasc Dis 2010:52:336–346.

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• Fig. 1. 

  Lymph node from a patient with sarcoidosis showing numerous nonnecrotizing granulomas typical of sarcoidosis. Hematoxylin and eosin stain. Original magnification, ×40. Courtesy of Dr Robert Padera, Brigham and Women's Hospital, Boston, Mass.

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• Fig. 2. 

  Gross specimen of a heart from a patient with cardiac sarcoidosis explanted at the time of cardiac transplantation. The ventricles are dilated, and there are scattered white areas in the myocardium in a seemingly random distribution, which represent scar tissue typical of cardiac sarcoidosis. Courtesy of Dr Robert Padera, Brigham and Women's Hospital, Boston, Mass.

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• Fig. 3. 

  Example of a myocardial granuloma, diagnostic of cardiac sarcoidosis. There is associated fibrosis. Hematoxylin and eosin stain. Original magnification, ×100. Courtesy of Dr Robert Padera, Brigham and Women's Hospital, Boston, Mass.

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• Fig. 6. 

  A, Combined resting PET scan using rubidium-82 and ¹⁸F-FDG scanning in a 53-year-old man with a history of pulmonary sarcoidosis who presented with palpitations and atrial flutter. From the top, each pair of images represents the rubidium-82 scan and, underneath it, the corresponding ¹⁸F-FDG image. The scans show a basal and midanteroseptal perfusion defect with intense FDG uptake in these regions suggestive of myocardial inflammation. Normal myocardium has no FDG uptake because it is using free fatty acids. B, CT images in the same patient demonstrating the intense cardiac uptake (left panel: combined PET-CT scan). Courtesy of Dr Sharmila Dorbala, Brigham and Women's Hospital, Boston, Mass.